Objective: The loss of liver glycine N-methyltransferase (GNMT) promotes liver steatosis and the transition to hepatocellular carcinoma (HCC). Previous work showed endogenous glucose production is reduced in GNMT-null mice with gluconeogenic precursors being used in alternative biosynthetic pathways that utilize methyl donors and are linked to tumorigenesis. This metabolic programming... https://hlarfszwqj2up.dsiblogger.com/67920465/implementation-of-an-animal-sporotrichosis-surveillance-and-control-program-southeastern-brazil